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Subsequent work by Sun and colleagues in the APP/PS1 mouse model identified downstream PI3K/AKT pathway activation as a major component of the mechanism, with the PI3K inhibitor wortmannin reversing Dihexas anti-inflammatory and anti-apoptotic effects [5]

-amyloid: the known unknowns

A heavy stable isotope-labelled GS*-NEM was used as an internal standard in the processed samples to achieve accurate quantitation and data normalisation (Figs

This suggests that SLC7A11 not only plays a critical role in protein and GSH synthesis through cysteine uptake but also dictates glutamine dependence 61

James et al., 2015) to increase the risk of ferroptosis